Of course. Familial syndromes, which are hereditary gene defects in the field of cancer genetics, can increase a person's risk of developing cancer. Most importantly, genetic variation that assists in tumor growth and initiation may impair the critical systems that control cell proliferation, differentiation, and apoptosis.
Instead of seeing inherited cancer as a disability, it is an opening that points out many precious processes in the cells and the beginning of carcinogenesis.
Being able to map the genes that could lead to high cancer risk across individuals and figure out their impact on cancer development can result in cancer biology being resolved as a complex tapestry.
Moreover, it is appropriate to comprehend the intricate relationship among environmental elements and the inherited genetic attribute leading to the development of cancer; how cancer develops is not well understood yet.
The risk of getting cancer produced as a result of irradiation exposure and the presence of mutations that may have arisen naturally over the course of time are often factors that predispose people to cancer development.
Whatever it takes to fully understand the multifactorial causes of cancer, whatever they are, genetic and environmental, is the baseline for designing clinically applicable treatments.
Through tailoring assessment, monitoring, and preventive steps for every patient, healthcare providers can, first of all, provide excellent management of cancer that is also affordable to society. By employing data-driven capabilities, accomplishing the outlined goal becomes achievable.
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